There are several common types of eyelid malpositions. This is a general medical term for eyelids that don’t sit and do what they are supposed to do. Normally the eyelids are firmly held against the eye. Tears lubricate the eyelid movements and the eyelids themselves function like the windshield wipers of the eyes. This is the result of a delicate balancing act between the eyelid position and tension of the eyelid ligaments. In the lower eyelid the muscle that helps close the eyelids functions like a muscular hammock to hold the lower eyelid against the globe.
In the upper eyelid the levator palpebra superioris muscle is responsible for opening the eyes by elevating the upper eyelids. The action of this muscle is transmitted to the upper eyelid by a broad fan like tissue called the levator aponeurosis. Individuals may be born with a droopy eyelid and the levator muscle may not work as effectively as a normal muscle. Under the microscope, the muscle may look incompletely developed with fat and connective tissue replacing the muscle that should be present. Crowell Beard, M.D, has referred to this as “developmental dystrophy”. As the levator muscle does not effectively lift the lid, there may also be insufficient traction on the skin by the same muscle to generate a crease in the affected upper eyelid. It is not uncommon to see a heavy eyelid and an absent eyelid crease. Occasionally the heavy eyelid is associated with other eyelid abnormalities such as the blepharophimosis syndrome. This syndrome has a heredity basis and can run in families. A less common form of congenital ptosis called Marcus-Gunn jaw-winking ptosis: movement of the jaw causes a fall in the position of one of the eyelids. Treatment of these conditions is based on the degree the levator muscle is able to lift the eyelid. This measure is called the levator function.
In adults, congenital ptosis is seen but the most common cause of upper eyelid ptosis in the adult is acquired ptosis. There are several causes of acquired ptosis. The most common cause appears to be attenuation of the levator aponeurosis, which becomes stretched out over time. This is also referred to as levator dehiscence ptosis but it is controversial if the levator actually “dehisces,” a medical term for separates, or if the attenuated tendon gets cut in the process of performing the surgical repair. Other causes of acquired ptosis include four broad categories: neurogenic, myogenic, traumatic, and mechanical. It is helpful to classify and properly diagnosis the basis of the droopy eyelid because this has a bearing on the choice for repairing the upper eyelid ptosis.
There are two principle approaches to repairing acquired upper eyelid ptosis. For small amounts of ptosis (1-2 mm) a simple test is performed during the preoperative assessment. An eye drop is instilled into the eyes and the response to the drops is measured. If the drop raises the eyelids to the desired height, the lid position is very likely to respond to a surgery referred to as a Conjunctival Muellerectomy. This surgery is performed from behind the upper eyelid using a special clamp called a Putterman ptosis clamp. If the eyelid does not respond to the eye drops or if the degree of ptosis is too large a second type of ptosis surgery is indicated. This type of surgery is called an anterior levator resection ptosis surgery. This surgery is performed through an incision at the eyelid crease on the outside of the eyelid. The levator aponeurosis is dissected and sutures are tied to effectively shorten the aponeurosis. Surgery is performed under local anesthesia. This facilitates repositioning of the eyelid. The patient is asked to open and close the eye. The sutures can be repositioned to adjust the eyelid position. Once this is satisfactory, the sutures are permanently tied and the eyelid is closed. Skin sutures are removed within a week. Swelling and bruising usually resolves in 10-14 days. A personal consultation with Dr. Steinsapir will determine if you are a candidate for upper eyelid ptosis surgery.
Laxity of the lower eyelid can present as either a turning in or turning out of the lower eyelid. These malpositions usually occur in older individuals. The type of lower eyelid malposition is determined by the nature of which structure have become weak in the eyelid. When the lower eyelid retractors become attenuated, the forces on the eyelid tend to favor an inward rotation of the lower eyelid. This is called entropion. This causes the eyelashes to rub against the eye and cause severe irritation. There are various approaches to address these issues. Typically the lower eyelid retractors are reattached and the lower eyelid is shortened. When the lower eyelid is lax and there is contraction of the lower eyelid skin or weakening of the orbicularis oculi muscle as occurs with Bell’s palsy, the lower eyelid rotates away from the eye. This is called ectropion. In this situation, the lower eyelid does not rub against the eye. However, the lid does not protect the eye and this can also cause irritation. Surgery is directed at lengthening the lower eyelid and resuspending the lid back against the globe. In both cases, an assessment of the midface support is essential. With entropion and ectropion, failure to address midface ptosis can lead to an unsatisfactory result. Swelling and bruising usually resolves in 10-14 days. A personal consultation with Dr. Steinsapir will determine if you are a candidate for repair of entropion or ectropion.